miR-483-5p offsets functional and behavioural effects of stress in male mice through synapse-targeted repression of Pgap2 in the basolateral amygdala – Nature Communications#stress Latest research alert! This is research in it’s earliest stages and it’s a very complex biotechnology/neuro pharmacology article but it’s considered a breakthrough. The reason it’s considered a breakthrough is because right now we’re not great at managing anxiety with medications. Would it be better if we could manage it with behavior and meditation/lower stress – of course. But, it helps to identify the areas and mechanisms that “relay emotionally relevant neural signals to the downstream brain centres responsible for the expression of behavioural reactions (e.g. risk avoidance, low food intake, social avoidance, increase in blood pressure and heart rate) that collectively assemble anxiety-like states7,8,10.” It’s important to remember that many mental health disorders are similar to physical diagnosis like asthma or type 1 diabetes. The “disease” is an identification of cells in the body, including the brain not producing the “expected” or “normal” amount of a substance we need to live our best lives. That’s why medicine and medications are introduced. The goal is always to help the individual achieve lasting wellness.
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